Oral Steroids

Anavar 10 mg Omega Meds

Omega Meds

110,00

In Stock (100 available)

Anavar 10 mg Omega Meds (10 mg) — Oxandrolone stands apart as the mildest and most therapeutically forgiving anabolic steroid in clinical and athletic use. Its exce…

100 in stock

This product is for laboratory research use only. Not for human consumption.

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Description

Oxandrolone (Anavar), Omega Meds, 10 mg. The most-tolerated oral anabolic across safety-profile measures. One of the few AAS with reasonable female protocols (5–20 mg/day). Stacks with virtually anything; bridge-compound favourite.

Key Benefits

  • Highest anabolic:androgenic ratio of any common AAS (~322–630 / 24)
  • Does NOT aromatise — zero oestrogenic side-effects
  • Minimally hepatotoxic relative to other 17α-alkylated orals
  • Mitochondrial fatty-acid uptake enhancement — cutting utility
  • One of few AAS with documented female protocols (5–20 mg/day)
  • Each unit dosed at 10 mg — see Recommended Dosage below for protocol-specific intake

Recommended Dosage

Research dosing: men 20–80 mg/day split two or three times daily; women 5–20 mg/day single dose. Standard cycle 6–8 weeks. Above 80 mg/day gains plateau while liver-enzyme burden continues rising.

How It Works

17α-alkylated DHT-derivative with a modified A-ring that strips androgenic expression while keeping anabolic activity. Does not aromatise. Binds the androgen receptor with modest affinity; results come from elevated protein synthesis, mitochondrial fatty-acid uptake, and erythropoiesis rather than raw AR occupation.

Pharmacokinetics

Oral bioavailability ~97% — highest of any AAS. Plasma half-life approximately 9 hours; splitting the daily dose across 2–3 administrations keeps serum levels flat. Hepatotoxicity low relative to methylated orals but still real; pull ALT/AST at week 4.

Potential Side Effects

Lipid panel disruption (HDL drop) is consistent. Mild LH/FSH suppression even at low doses — mini-PCT appropriate after 6+ week cycles. No water retention, no oestrogenic effects. Androgenic sides (hair-thinning in susceptible users) possible at higher doses.

Cycle & Stacking Guide

Cutting adjunct on a testosterone base, 6–8 weeks. Stacks with test + masteron for pre-contest hardening or with clen + T3 for recomp. Often used as a finisher (final 6 weeks of a 14-week cycle). TUDCA 500 mg/day throughout.

Manufacturer Notes

Omega Meds focuses on injectable blends with high-concentration formulations. Batch verification available through their distributor network.

Storage & Handling

Store in the original blister or bottle at 15–25 °C, away from direct sunlight, heat, and humidity. Oral preparations lose potency faster if exposed to moisture; keep the desiccant (if included) with the tablets. Keep out of reach of children. For research and educational purposes only.

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Oxandrolone

Physical & Chemical Properties for Research Purposes
Chemical structure of Oxandrolone (C19H30O3) for laboratory analysis
2D structural representation · PubChem CID 5878 ↗
Chemical Identity
# CAS Registry Number 53-39-4
Σ IUPAC Name (1S,2S,7S,10R,11S,14S,15S)-2,15-dimethyl-5-oxotetracyclo[8.7.0.0^{2,7}.0^{11,15}]heptadecan-14-yl acetate
F Molecular Formula C19H30O3
M Molecular Weight 306.44 g/mol
SMILES C[C@@H]1C[C@H]2[C@@H]3CC[C@](O)(C(C)=O)[C@@]3(C)CC[C@@H]2[C@@]4(C)C=CC(=O)C[C@@H]14
InChIKey QSLJIVKCVHQPLV-PEMPUTJUSA-N
Melting Point 235-238 °C
Solubility Slightly soluble in water; soluble in acetone, chloroform
Biological Half-life 9 hours (oral)
PubChem CID 5878 ↗
Pharmacological Profile
Anabolic Rating 322
Androgenic Rating 24
Aromatization None
Hepatotoxicity Low
Detection Time 3 weeks

Clinical Notes

DHT-derived oral with an oxygen atom substituting C-2 on the A-ring — a modification that simultaneously protects the molecule from hepatic metabolism and neutralises 5alpha-reductase affinity. Clinical FDA indications (HIV wasting, severe burn recovery, Turner syndrome) were approved on the strength of its anabolic-to-androgenic dissociation, which is the widest in the oral AAS class. Aromatisation is mechanistically impossible. Hepatotoxicity is real but mildest of C17alpha-alkylated compounds — ALT typically rises 1.5–2.5x ULN at 40 mg/day x 8 weeks. Lipid profile degradation is severe — HDL suppression 50%+ is reproducible. Effect at 20–40 mg/day: strength gain without water retention, minimal scale weight change (5–8 kg lean mass maximum), visible muscle hardening.

Known trade names: Anavar, Oxandrin, Lonavar

Data sourced from published pharmacological literature and authoritative chemical databases (PubChem, DrugBank, ChEBI). Provided for identification and research reference only.